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The Ayurvedic drug Ksheerabala (101) ameliorates alcohol-induced neurotoxicity by down-regulating the expression of transcription factor (NFkB) in rat brain

| Posted in: Science

Journal name: AYU (Journal of Research in Ayurveda)
Original article title: The Ayurvedic drug Ksheerabala (101) ameliorates alcohol-induced neurotoxicity by down-regulating the expression of transcription factor (NFkB) in rat brain
AYU is an internationally recognized quarterly journal dedicated to advancing research in Ayurveda. The journal covers a wide range of topics, including clinical and pharmacological research in Ayurveda's eight branches, herbal remedies, phytochemistry, and ethnomedicine.

Original source:

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Author(s):

S. Rejitha
P. Prathibha
Indira Madambath


AYU (Journal of Research in Ayurveda):

(An International Quarterly Journal of Research in Ayurveda)

Full text available for: The Ayurvedic drug Ksheerabala (101) ameliorates alcohol-induced neurotoxicity by down-regulating the expression of transcription factor (NFkB) in rat brain

Year: 2015 | Doi: 10.4103/0974-8520.182749

Copyright (license): CC BY-NC-SA 4.0


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Summary of article contents:

Introduction: Most of the pharmaceutical effects of alcohol are due to its accumulation in the brain. Ksheerabala (101) an Ayurvedic formulation mainly used against central nervous system disorders.

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Discover the significance of concepts within the article: �The Ayurvedic drug Ksheerabala (101) ameliorates alcohol-induced neurotoxicity by down-regulating the expression of transcription factor (NFkB) in rat brain�. Further sources in the context of Science might help you critically compare this page with similair documents:

Ayurvedic formulation, Oxidative stress, Reactive oxygen species, Cognitive dysfunction, Neuroprotective properties, Animal Ethics Committee, Oxidative damage, Ayurvedic Drug, Neuroinflammation, Glutathione (GSH), Experimental period, Histopathology, Ksheerabala (101), Statistical program, Glial cells, Proinflammatory response, Total RNA isolation, Biochemical evaluation, Antioxidant enzyme, Protein carbonyl, Central nervous system disorder, Male Albino Rat, Inflammatory marker, Lipid peroxidation product.

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